Bio-compatibility of heat exchanger components and temperature effects on formed elements and plasma proteins

 

 

    The heat transfer surface is usually made of stainless steel or aluminium and rarely plastic

     Both aluminium & stainless steel have good thermal conductivity and are readily coated with polymers to minimise blood interactions

     However with long term extra corporeal life support, inorganic emboli related to an aluminium heat exchanger (no longer sold) have been found in the brain. Problems with corrosion in aluminium heat exchangers resulting in blood leaks and circuit contamination have resulted in the preferential use of stainless steel

     Intravascular aggregation has been observed at rapid cooling rates

     Blood damage in the form of protein denaturation occurs at temperatures greater than 43°C

 

        Protein molecules are large and complex. Their activity depends on their shape and chemical composition. The denaturing or inactivating, of proteins involves the changing of their shape &/or breaking off attached chemical groups. Denatured enzymes may loose their catalytic properties, the anti-body-antigen mechanism may be altered, hormones may loose their function etc.. Surface electrical charges on these molecules are altered, they become sticky and form aggregates. Soluble proteins may become insoluble and precipitate. Denatured globulins may cryoprecipitate (ppt when exposed to hypothermia). Lipid components freed from lipoproteins coalesce to form fat emboli. Some proteins become more active resulting in complement and platelet activation.